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For the hundreds of millions of people presently infected with malaria, survival may depend on relatively non-specific immune effector mechanisms. Progress has been made in understanding the anti-parasitic properties of tumor necrosis factor-alpha, interferon-gamma and nitric oxide, in defining the parasite toxins that induce tumor necrosis factor-alpha production, and in exploring the role of cytokines and adhesion molecules in the pathogenesis of cerebral malaria.

Original publication

DOI

10.1016/s0952-7915(06)80034-8

Type

Other

Publication Date

08/1992

Volume

4

Pages

425 - 431

Addresses

Institute of Molecular Medicine, John Radcliffe Hospital, Oxford, UK.

Keywords

Liver, Erythrocytes, Animals, Humans, Plasmodium falciparum, Malaria, Cerebral, Malaria, Malaria, Falciparum, Tumor Necrosis Factor-alpha, Protozoan Vaccines, Immunity, Host-Parasite Interactions